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Enter orf / gene / drug:
(e.g. pex22 or lovastatin)
YOL164W / BDS1
Section 1: Phenotypes.
Gene-drug relationships for YOL164W
(Hillenmeyer et al., Science 2008)
The top 10 experiments that caused a fitness defect in the YOL164W deletion mutants. On the left is a table listing the top 10 conditions inducing sensitivity; on the right is a visualization of those conditions. Edges may appear more than once if the condition was tested multiple times.
We generally recommend using a significance cutoff of p-value < 0.01. For the "phenotype for all genes" analysis, we used p-value < 1e-5 to control for multiple hypothesis testing. For details, see
The Chemical Genomic Portrait of Yeast
[No Heterozygous results.]
[No Homozygous results.]
Section 2: Co-fitness interactions.
Gene-gene relationships for YOL164W
(Hillenmeyer et al., Genome Biol. 2010)
This study extends the preliminary analysis of clustering genes according to co-fitness across experiments (Hillenmeyer et al., Science 2008). Here, instead of clustering genes, we analyzed the co-fitness between all pairs of genes, testing several types of correlation.
Heterozygous co-fitness interactions
[No heterozygous results.]
Homozygous co-fitness interactions
[No homozygous results.]
External information about YOL164W
Visit YOL164W in [
Previously known GO terms:
dodecyl sulfate metabolism
alkyl sulfatase activity
cellular component unknown
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