|Patient: 56 year old male|
R/O disc herniation.
MRI of the lumbar spine reveals the following:
L5-S1: There is mild signal loss of the nucleus pulposus without loss of disc height. Dorsal annular bulging is noted with a curvilinear high intensity zone signal abnormality at the 7:00 position consistent with a partial peripheral annular tear/fissure effacing the traversing right S1 nerve root with mild dorsal displacement (T2 axial image 19, sagittal image 7). In addition, there is mild facet hypertrophy.
L4-L5: There is bilateral spondylolysis with Grade II spondylolisthesis. Advanced degeneration and marked disc space narrowing of the intervertebral disc is noted with Modic Type I and II reactive end plate marrow changes. There is associated facet hypertrophy. There is prominent dorsal and cephalad annular bulging secondary to the spondylolisthesis resulting in lateral recess and neuroforaminal stenosis. Clinical correlation advised for associated radicular involvement.
L3-L4: The intervertebral disc is unremarkable. There is mild facet hypertrophy with small effusions noted in the zygapophyseal joints.
L2-L3: The intervertebral disc has a normal appearance. The spinal canal, neural foramina and contents are well maintained. The posterior elements are unremarkable.
L1-L2: Moderate signal loss of the nucleus pulposus with mild loss of disc height. Anterior and posterior discal bulging measuring 2-3 mm is identified, most pronounced anteriorly with associated end plate spondylosis. There is compression of the thecal sac but no neurological compromise.
T12-L1: The intervertebral disc has a normal appearance. The spinal canal, neural foramina and contents are well maintained. The posterior elements are unremarkable.
There are no compression fractures or marrow lesions within the vertebral bodies. The conus medullaris does not show any intrinsic or extrinsic abnormality. The paraspinal musculature shows mild atrophy and asymmetry in the lumbosacral junction beginning at the level of the spondylolysis and spondylolisthesis of L4-L5. Specifically, there is hypoplasia of the multifidi, right greater than left L4-L5 and L5-S1. Retroperitoneal structures visualized in this examination are unremarkable.
REFERRING PHYSICIAN: DR. SALIMSH CUMBER, MD
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Case Number: 3831656Last Updated: 01-24-2007 The reader is fully responsible for confirming the accuracy of this content.
The reader is fully responsible for confirming the accuracy of this content.