uprightdoc wrote:I have never heard of the "thirteen circuits" of the body. Connecting the teeth to the sacroiliac joints is ridiculous. What I am discussing here is definitely based on science and it's far from being just my opinion. Although the theory may be weak, CCSVI is also based on science. Unlike medical doctors I am not suggesting that we write laws to prevent MS patients from seeking alternative treatments. I think MS patients should be allowed to try anything they want as long as it's not harmful such as TMJ, orthotics, chelation therapy, colonics, ionized water, crystals, magnets, polarized glasses, color therapy, Bach Flower Remedies, grounding, sleeping under pyramids, fecal transplants, etc.
MrSuccess wrote:...Are you in agreement in regard to the fragile state that the brain is in and the effects on it when bloodflow IN is compromised ?...
NZer1 wrote:... In the 1960s, a neurosurgeon in Bogota, Columbia made the controversial claim that he could reverse neurodegeneration by surgically diverting an excess of cerebrospinal fluid (CSF) in the brains of his patients with a shunt.
Professor Salomon Hakim first published his thesis in 1964 and then published 6 case reports of "normal pressure hydrocephalus" in The New England Journal of Medicine and the Journal of the Neurological Sciences in 1965. Hakim rose to the forefront of academic medicine as he described a newfound ability to reverse symptoms of “neurodegeneration” that had long been considered irreversible.
It is important to understand that today, 60 years after Hakim's discovery, treating normal pressure hydrocephalus is an accepted practice, even though diagnosing NPH is an inexact science. There were never any double blinded clinical trials for this surgery. Hakim claimed he could reverse gait impairment, cognitive problems and urinary incontinence by diverting CSF flow. And the proof was in his patients' recovery. No one cries "placebo effect!" after a patient recovers mobility, cognition or bladder control once treated for NPH...
The association between EBV infection and CCSVI has not yet been explored; however, it could be hypothesized that venous stasis in the superior saggital sinus due to extracranial outflow impairment could affect the drainage of bridging veins that pass through the subarachnoid space (near the meninges and EBV-infected B-cell follicles) and contribute to EBV activation. The venous stasis hypothesis in the SSS may contribute to understanding why so many different viruses and bacteria [3,111] have been linked to increased MS susceptibility risk over the last 50 years.
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